Registration Dossier

Data platform availability banner - registered substances factsheets

Please be aware that this old REACH registration data factsheet is no longer maintained; it remains frozen as of 19th May 2023.

The new ECHA CHEM database has been released by ECHA, and it now contains all REACH registration data. There are more details on the transition of ECHA's published data to ECHA CHEM here.

Diss Factsheets

Administrative data

Hazard for aquatic organisms

Freshwater

Hazard assessment conclusion:
PNEC aqua (freshwater)
PNEC value:
76.7 µg/L
Assessment factor:
1
Extrapolation method:
sensitivity distribution
PNEC freshwater (intermittent releases):
76.7 µg/L

Marine water

Hazard assessment conclusion:
PNEC aqua (marine water)
PNEC value:
22.7 µg/L
Assessment factor:
1
Extrapolation method:
sensitivity distribution
PNEC marine water (intermittent releases):
22.7 µg/L

STP

Hazard assessment conclusion:
PNEC STP
PNEC value:
193.6 µg/L
Assessment factor:
1
Extrapolation method:
sensitivity distribution

Sediment (freshwater)

Hazard assessment conclusion:
PNEC sediment (freshwater)
PNEC value:
438.6 mg/kg sediment dw
Assessment factor:
1
Extrapolation method:
sensitivity distribution

Sediment (marine water)

Hazard assessment conclusion:
PNEC sediment (marine water)
PNEC value:
210.3 mg/kg sediment dw
Assessment factor:
1
Extrapolation method:
sensitivity distribution

Hazard for air

Air

Hazard assessment conclusion:
no hazard identified

Hazard for terrestrial organisms

Soil

Hazard assessment conclusion:
PNEC soil
PNEC value:
132.5 mg/kg soil dw
Assessment factor:
1
Extrapolation method:
sensitivity distribution

Hazard for predators

Secondary poisoning

Hazard assessment conclusion:
no potential for bioaccumulation

Additional information

The ecotoxicology of zinc lactate can be understood in terms of the ecotoxicology of lactic acid/lactates and the ecotoxicology of zinc(II). The PNECs derived from the Zn(II) data are adequately protective for zinc lactate when compared to the information available for zinc lactate itself.

Lactic acid is a ubiquitous and essential molecule of life; acute effects, as well as (external) long-term effects are most likely the results of physical processes (pH-effect; electrostatic effects). Chronic effects are not relevant in the context of risk assessment. PNECs are derived based on the acute effects. PNECs are below the chronic no-effect levels, where available.

From the zinc chloride dossier:

A basic assumption made in this hazard assessment and throughout this CSR, (in accordance to the same assumption made in the EU RA process) is that the ecotoxicity of zinc and zinc compounds is due to the Zn++ ion. As a consequence, all aquatic, sediment and terrestrial toxicity data in this report are expressed as “zinc”, not as the test compound as such, because ionic zinc is considered to be the causative factor for toxicity. A further consequence of this is that all ecotoxicity data obtained on different zinc compounds, are mutually relevant for each other. For that reason, the available ecotoxicity databases related to zinc and the different zinc compounds are combined before calculating the PNECs. The only way zinc compounds can differ in this respect is in their capacity to release zinc ions into (environmental) solution. That effect is checked eventually in the transformation/dissolution tests and may result in different classifications.

Conclusion on classification

Based on the relatively high aquatic toxicity of zinc - Zn(II) - classification of zinc dilactate as "Aquatic Acute 1" and "Aquatic Chronic 3" is warranted.