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Diss Factsheets
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EC number: 244-334-7 | CAS number: 21324-40-3
- Life Cycle description
- Uses advised against
- Endpoint summary
- Appearance / physical state / colour
- Melting point / freezing point
- Boiling point
- Density
- Particle size distribution (Granulometry)
- Vapour pressure
- Partition coefficient
- Water solubility
- Solubility in organic solvents / fat solubility
- Surface tension
- Flash point
- Auto flammability
- Flammability
- Explosiveness
- Oxidising properties
- Oxidation reduction potential
- Stability in organic solvents and identity of relevant degradation products
- Storage stability and reactivity towards container material
- Stability: thermal, sunlight, metals
- pH
- Dissociation constant
- Viscosity
- Additional physico-chemical information
- Additional physico-chemical properties of nanomaterials
- Nanomaterial agglomeration / aggregation
- Nanomaterial crystalline phase
- Nanomaterial crystallite and grain size
- Nanomaterial aspect ratio / shape
- Nanomaterial specific surface area
- Nanomaterial Zeta potential
- Nanomaterial surface chemistry
- Nanomaterial dustiness
- Nanomaterial porosity
- Nanomaterial pour density
- Nanomaterial photocatalytic activity
- Nanomaterial radical formation potential
- Nanomaterial catalytic activity
- Endpoint summary
- Stability
- Biodegradation
- Bioaccumulation
- Transport and distribution
- Environmental data
- Additional information on environmental fate and behaviour
- Ecotoxicological Summary
- Aquatic toxicity
- Endpoint summary
- Short-term toxicity to fish
- Long-term toxicity to fish
- Short-term toxicity to aquatic invertebrates
- Long-term toxicity to aquatic invertebrates
- Toxicity to aquatic algae and cyanobacteria
- Toxicity to aquatic plants other than algae
- Toxicity to microorganisms
- Endocrine disrupter testing in aquatic vertebrates – in vivo
- Toxicity to other aquatic organisms
- Sediment toxicity
- Terrestrial toxicity
- Biological effects monitoring
- Biotransformation and kinetics
- Additional ecotoxological information
- Toxicological Summary
- Toxicokinetics, metabolism and distribution
- Acute Toxicity
- Irritation / corrosion
- Sensitisation
- Repeated dose toxicity
- Genetic toxicity
- Carcinogenicity
- Toxicity to reproduction
- Specific investigations
- Exposure related observations in humans
- Toxic effects on livestock and pets
- Additional toxicological data
Toxicological Summary
- Administrative data
- Workers - Hazard via inhalation route
- Workers - Hazard via dermal route
- Workers - Hazard for the eyes
- Additional information - workers
- General Population - Hazard via inhalation route
- General Population - Hazard via dermal route
- General Population - Hazard via oral route
- General Population - Hazard for the eyes
- Additional information - General Population
Administrative data
Workers - Hazard via inhalation route
Systemic effects
Long term exposure
- Hazard assessment conclusion:
- DNEL (Derived No Effect Level)
- Value:
- 0.931 mg/m³
- Most sensitive endpoint:
- repeated dose toxicity
- Route of original study:
- Oral
DNEL related information
- Explanation for the modification of the dose descriptor starting point:
- Justification provided in "justification and comments" field below
Acute/short term exposure
- Hazard assessment conclusion:
- high hazard (no threshold derived)
- Most sensitive endpoint:
- skin irritation/corrosion
DNEL related information
Local effects
Long term exposure
- Hazard assessment conclusion:
- high hazard (no threshold derived)
- Most sensitive endpoint:
- skin irritation/corrosion
Acute/short term exposure
- Hazard assessment conclusion:
- high hazard (no threshold derived)
- Most sensitive endpoint:
- skin irritation/corrosion
DNEL related information
Workers - Hazard via dermal route
Systemic effects
Long term exposure
- Hazard assessment conclusion:
- DNEL (Derived No Effect Level)
- Value:
- 133 µg/kg bw/day
- Most sensitive endpoint:
- repeated dose toxicity
- Route of original study:
- Oral
DNEL related information
- Explanation for the modification of the dose descriptor starting point:
- Justification provided in "justification and comments" field below
Acute/short term exposure
- Hazard assessment conclusion:
- high hazard (no threshold derived)
- Most sensitive endpoint:
- skin irritation/corrosion
DNEL related information
Local effects
Long term exposure
- Hazard assessment conclusion:
- high hazard (no threshold derived)
- Most sensitive endpoint:
- skin irritation/corrosion
Acute/short term exposure
- Hazard assessment conclusion:
- high hazard (no threshold derived)
- Most sensitive endpoint:
- skin irritation/corrosion
Workers - Hazard for the eyes
Local effects
- Hazard assessment conclusion:
- high hazard (no threshold derived)
Additional information - workers
Calculation of DNELs for LiPF6 exposure
[1] Short-term DNELs
The reactive and corrosive properties of LiPF6 make calculation of short-term DNELs inappropriate. Skin contact or inhalation of particulate material is avoided by the use of appropriate containment procedures and PPE, as detailed in the relevant Guidance on Safe Use.
[2] Long-term DNELs
The rapid hydrolysis of LiPF6 in contact with moisture, releasing HF, LiF and H3PO4 makes it appropriate to based long-term DNELs on the degradation products which could be absorbed and systemically distributed in the event of LiPF6 exposure. As discussed earlier, these degradants are HF, F-, Li+ and PO4-. Each is considered in turn.
In respect of HF: the IOELV of 1.5 mg/cu.m (as F-) would be appropriate as a long-term worker DNEL via inhalation, corresponding to 15 mg/day or 214 µg/kg/day.
In respect of F-: 100 µg/kg/day (7 mg/day) would be appropriate as a long-term worker DNEL via ingestion.
In respect of Li+: the human NOAEL of 14.3 µg/kg/day (1 mg/day) would be appropriate as a long-term worker DNEL via inhalation (and data from human therapeutic use demonstrate that a much higher oral dosage is well tolerated) .
In respect of PO4: the MTDI of 70 mg/kg/day (as P) or the lower ADI of 30 mg/kg/day would be appropriate as a long-term worker DNEL via ingestion.
Based on molecular weight, hydrolysis of 152 mg LiPF6 will release 7 mg Li+, 31 mg P, 100 mg HF and 114 mg F-. From this we can see that:
- for HF, 214 µg F-/kg/day corresponds to 325 µg LiPF6/kg/day (by inhalation)
- for F-, 100 µg/kg/day corresponds to 133 µg LiPF6/kg/day (by ingestion)
- for Li+, 14.3 µg/kg/day corresponds to 313 µg Li PF6/kg/day (by inhalation)
- for PO4, 70 mg P/kg/day corresponds to 343 mg LiPF6/kg/day; the lower ADI (unconditional) value of 30 mg P/kg/day corresponds to 147 mg LiPF6/kg/day (by ingestion).
It is therefore evident that the fluoride released as LiPF6 enters biological systems is the most toxic component, and provides the toxicity marker for long-term worker DNEL determination. Since this is readily absorbed both by skin contact and from the gastrointestinal tract, it is appropriate to apply an equal DNEL value for dermal and oral routes. In addition, since it is the toxicity of F- which causes much of the toxicity observed following HF exposure there is no reason to suppose that exposure by inhalation will elicit lesser toxicity than oral or dermal exposure, so to apply the precautionary principle the inhalation DNEL is reduced to equal that for ingestion or dermal contact. The following long-term worker DNEL values are therefore set for LiPF6:
- for inhalation exposure, 133 µg/kg/day (0.931 mg/m3)
- for dermal contact, 133 µg/kg/dayGeneral Population - Hazard via inhalation route
Systemic effects
Long term exposure
- Hazard assessment conclusion:
- no hazard identified
Acute/short term exposure
- Hazard assessment conclusion:
- no hazard identified
DNEL related information
Local effects
Long term exposure
- Hazard assessment conclusion:
- no hazard identified
Acute/short term exposure
- Hazard assessment conclusion:
- no hazard identified
DNEL related information
General Population - Hazard via dermal route
Systemic effects
Long term exposure
- Hazard assessment conclusion:
- no hazard identified
Acute/short term exposure
- Hazard assessment conclusion:
- no hazard identified
DNEL related information
Local effects
Long term exposure
- Hazard assessment conclusion:
- no hazard identified
Acute/short term exposure
- Hazard assessment conclusion:
- no hazard identified
General Population - Hazard via oral route
Systemic effects
Long term exposure
- Hazard assessment conclusion:
- no hazard identified
Acute/short term exposure
- Hazard assessment conclusion:
- no hazard identified
DNEL related information
General Population - Hazard for the eyes
Local effects
- Hazard assessment conclusion:
- no hazard identified
Additional information - General Population
General population DNELs have not been derived as no consumer exposure to the substance is expected. Hazard assessment conclusion for the general population "no hazard identified" has been selected based on the fact that no exposure is expected to occur.
Information on Registered Substances comes from registration dossiers which have been assigned a registration number. The assignment of a registration number does however not guarantee that the information in the dossier is correct or that the dossier is compliant with Regulation (EC) No 1907/2006 (the REACH Regulation). This information has not been reviewed or verified by the Agency or any other authority. The content is subject to change without prior notice.
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