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EC number: 237-529-3
CAS number: 13826-66-9
The aquatic toxicity of zirconium dinitrate oxide (a 'water soluble'
zirconium compound) is evaluated using data for zirconium dinitrate
oxide as well as data from read across substances such as zirconium
acetate and zirconium dichloride oxide (other 'water soluble' zirconium
compounds which behave similarly in aqueous media), zirconium basic
carbonate (a sparingly soluble zirconium compound), and insoluble
zirconium compounds such as zirconium dioxide and a reaction mass of
cerium dioxide and zirconium dioxide. Read across from insoluble or
sparingly soluble zirconium substances is considered acceptable because
several experiments (e.g., water solubility study and study on
pH-dependency of water solubility (Bauer, 2015), and an acute toxicity
to fish study (Sipos, 2015)) indicated that zirconium from the 'water
soluble' zirconium dinitrate oxide does not stay in solution at
environmentally relevant pH levels. Rapid hydrolysis occurs, resulting
in precipitation of zirconium hydroxides or zirconium dioxide. Further,
heavy complexing will occur with phosphates, which results in 100%
precipitation whenever phosphate is present in excess. Complexation with
carbonates may also result in progressive precipitation with increasing
pH levels. As a result, at environmentally relevant conditions,
zirconium dinitrate oxide cannot be considered as a water soluble
compound. Overall, zirconium is not bioavailable in the aquatic
environment and therefore no adverse effects are to be expected on
Further argumentation for the read across approach is given in the read
across document attached to IUCLID Section 13.
Zirconium dinitrate oxide is concluded not to be harmful or toxic
(acutely) to fish and aquatic invertebrates. The studies that were used
for endpoint coverage (performed with zirconium dinitrate oxide and/or
read across substances) were performed according to internationally
accepted guidelines and did not report any acute adverse effects
(mortality in fish, immobilisation in daphnids) at the limit test
concentration of 100 mg/L. In algal growth experiments with read across
substances, it was demonstrated that the observed growth inhibition was
concurrent with phosphate depletion. Toxicity to algae is therefore due
to phosphate deprivation as no primary toxic effects have been observed.
Phosphate deprivation is a secondary effect which is not considered
relevant at a normal environmental scale. Therefore, no effects on algae
are expected in the environment either. Finally, zirconium dinitrate
oxide was concluded not to be toxic to aquatic microorganisms based on
the results of an activated sludge respiration inhibition test performed
with the read across substance zirconium acetate.
Information on Registered Substances comes from registration dossiers which have been assigned a registration number. The assignment of a registration number does however not guarantee that the information in the dossier is correct or that the dossier is compliant with Regulation (EC) No 1907/2006 (the REACH Regulation). This information has not been reviewed or verified by the Agency or any other authority. The content is subject to change without prior notice.Reproduction or further distribution of this information may be subject to copyright protection. Use of the information without obtaining the permission from the owner(s) of the respective information might violate the rights of the owner.
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